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Tendons are chewie

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ktepps's version from 2016-11-19 15:02

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Question Answer
tenoblast arespecialized fibroblasts
tenocytes specialized fibrocytes, that make collagen I the main protein of the tendons extracellular matrix
how can you reveal the synchronously kinked arachiture of tendonspolarized light
common tendon injury of horsesDDFT, SDFT, suspensory ligament, DDFT/navicular bone/navicular dz
common tendon issues of dogcranial cruciate ligament, common calcaneal tendon
common tendon issue in ruminantslower limb extensor/flexor tendons
how inflammatory is tendinitisbiopsy specimens often demonstrate the presence of degenerative changes with absent or minimal inflammation
tendon injury is thought to occur through a traumatic mechanism, atraumatic, idiopathic degenerative tendonopathy is thought to occur as well
in the absense of inflammation what degenerative changes are seen in tendonsadvanced and extensive chondroid metaplasia, osseous metaplasia, fibroplasia/fibrosis and neovascularization
idiopathic degenerative injury occurs innavicular disease of horses
Traumatic tendon injury, also known as a strain, occur in what two waysacute/catostrophic injury or chronic/repetitive injury
actue/catostrophic injuryis the result of dramatic physical forces acting on the tendon at a single time(open and closed)
what is an open traumalacerations
what is a closed traumatendon ruptures as a result of high impact forces
infection of the tendon sheath istenosynovitis
chronic repetitive injuries are often seen in what parts of the equinrDDFT and SDFT
Tendon function is not compromised if the deformation is~4% or less
if stretch exceeds what % there is failure~8%
CS of tendon injurygrossly swollen, edematous, hot and painful (bowed tendon)
what is happening at the cellular level during traumatic injury of a tendonthe extracellular matrix and vascular supply of the tendon are disrupted. Hemorrhage, fibrin, inflammation and necrosis may be evident
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Question Answer
at the start of the repair stage, how does the tendon appearopaque tan to yellow and may be dramatically thickened (fibroplasias/granulation tissue)
at the early stages of repair, what is happening cellularlyhighly ordered parallel arrangement of tenocytes and extracellular matrix is lost and is replaced by a more chaotic, cellular (hyperplastic) fibroplasia, best seen in polarized light in
in fibrotic tendons, the predominant type I collagen of the extracellular matrix becomesintermixed with type III collagen, a fibrous protein less able to withstand tensile forces
in fibrotic tendons, the ordered, parallel arrays of tenocytes are replaced byhaphazard, densely and erratically distributed fibroblasts, resulting in weaker tendon
Degenerative suspensory desmitissunken pastern and/or fetlock joint
desmitisligamentous inflammation
desmopathysprain
cranial drawer sign indicatescomplete or partial rupture of the cranial cruciate ligament
Arthrogryposis"crooked joint"-the abnormal positioning of joints in terms of fixed overextesion or overflexion
where is the abnormality found in arthrogryposismay be in the tendon, but more often, the problem is myopathic or neurogenic (CNS)
proposed pathogenesis for arthrogryposis isfetal CNS lesion in utero --> lack of fetal movement --> fixation of the joints
possible plants that cause arthrogryposislupine species, poison hemlock, locoweed and/or tobacco
which virus infections cause arthrogryposisAkabane virus, Cache Valley virus, bluetongue virus, border disease virus, and Schmallenberg virus
how long is the remodling phase of healing a fibrously scarred tendonmonths to years in the remodeling stage
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