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ekadar's version from 2016-07-08 10:47

nuclei

Question Answer
mediates satietyventromedial
mediate hungerlateral
destruction leads to hyperphagiaventromedial
destruction leads to anorexialateral
mediates heat dissipation via parasympatheticsanterior
destruction leads to hyperthermiaanterior
mediates heat conservation via parasympatheticsposterior
destruction leads to hypothermiaposterior
secretion of dopamine, GH, and gonadotropinarcuate
ADH, CRH, oxytocin, TRHparaventricular
ADH and oxytoxcinsupraoptic
circadian rhythmsuprachiasmatic
stimulated by leptinventromedial
inhibited by leptinlateral
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neurotransmitters

Question Answer
NElocus cerelus (pons)
dopamineVTA and substania nigra pars compacta
serotoninraphe nucleus (pons, medulla, midbrain)
ACHbasal nucleus of meynert
GABAnucleus accumbens
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name duration of benzo

Question Answer
triazolamshort
alprazolamshort
oxazepamshort
estrazolamintermediate
temazepamintermediate
lorazepamintermediate
diazepamlong
flurazepamlong
chlordiazepoxidelong
chlordiazepoxidelong
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Regulation of Gluconeogenesis and Glycolysis

Question Answer
PFK-1 stimulated byfructose 2, 6 bisphosphatase
PFK-1 inhibited byATP, citrate
Fructose 1, 6 bisphosphatase does whatstep of gluconeogenesis
Fructose 1,6 bis to fructose 6-p what enzymefructose 1, 6 bisphosphatase
fructose 1,6 bisphosphatase inhibited byfructose 2,6 bisphos
What makes fructose 2, 6 bisphosphatasePFK-2 stimulated by insulin
what converts fructose 6, bisphosatase to fructose 6-pfructose 2, 6 bisphosphatase stimulated by glucagon
Takeway with insulin aroundmore fructose 2,6 is made and PFK-1 is activated and fructose 1,6 is inhibited
Takeway with glucagon aroundless fructose 2, 6 is made and gluconeogenesis is favored
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Cardio

Question Answer
most common cause of inhospital death due to MIcardiogenic shock/ventricular failure
LV free wall ruptures happens when3 to 7 days after onset of MI
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