tomi1's version from 2017-01-30 12:22

Section 1

Question Answer
define OAdegeneration joint disease-articular cartilage destruction
features of oastiffnes, pain, 'GIVING WAY', ROM, loss of function
which joints affectedknee, DIP hand, CMC thumb,hip, spine
which gender is more commonly affectedelderly women (menopause- loss of oestrogen- loss of inhibition on osteoclasts)
incidence20%=most common joint disorder
what is a RF for fallsosteoarthritis

Section 2

Question Answer
modifiable RFobesity,inactivity, occupation, HYPERMOBILE JOINTS
non-modifiable RFFH,gender,age, menopause,CRYSTAL ARTHOPATHY
primary causesidiopathic, FH
secondary causesmetabolic, anatomical,traumatic, inflammatory
metabolic causesgout, acromegaly,haemochromatosis
anatomical causes common in which age groupchildren - SUFE-Perthe's-CDH
traumaAVN,cruciate injury,

Section 3

Question Answer
pathology OAdestruction cartilage- changes in SC bone- debris + CK release into joint- inflammation SF- joint effusion
dd OA fibromyalgia, RA, gout, psuedogout
what time of day is pain worse and whyend of the day - mechanical pain - linked to movement
fibromyalgia vs oaROM maintained in fibromyalgia
RA vs OAlarger joints, swelling in RA

Section 4

Question Answer
investigations for OABLOODS + XR
bloods for OAFBC normal, inflammatory markers normal, RF-ve
XR for OA narrow joint space, subcondral sclerosis, osteophytes, cysts
osteophytesirregular bone outgrowth at joint margin
management in OAeducation-conservative-surgery
conservative managment OAMEDS + PHYSIO (strengthen muscles help to protect joint)
what analegsia should be avoided in OAnsaids- GU - instead consider paracetamol
what surgical options are availableosteotomy, joint replacement (hip or knee)
what injections are availableprednisolone, hyaluronon (natural component of SF)