Krebs Cycle

jonjonnavarro's version from 2015-10-14 04:59

Staphylococcus Virulence Factors

Inhibits chemotaxis and phagocytosis inhibits proliferation of mononuclear cells: Capsule
Facilitates adherence to foreign bodies: Capsule
Provides osmotic stability: Peptidoglycan
Stimulates production of endogenous pyrogen: Peptidoglycan
Endotoxin like activity: Peptidoglycan
Leukocyte chemoattractant Abscess formation: Peptidoglycan
Inhibits phagocytosis: Peptidoglycan
Regulates cationic concentration at cell membrane: Techoic Acid
Binds to fibronectin: Techoic Acid
Inhibits antibody-mediated clearance by binding IgG, IgG2 and IgG4 Fc receptors: Protein A
Leukocyte chemoattractant: Protein A
Anticomplementary: Protein A
Osmotic barrier: Cytoplasmic membrane
Regulates transport into and out of cell: Cytoplasmic membrane
Site of biosynthetic and respiratory enzymes: Cyoplasmic membrane
Toxic for many cells, including leukocytes, erythrocytes, macrophages, platelets and fibroblasts: Cytotoxin
Serine protease that split the intercellular bridges in the stratum granulosum epidermis: Exfoliative toxins
Superantigens stimulates proliferation of T cells and release of cytokines: Enterotoxins
Stimulates release of inflammatory mediators in mast cells, increasing intestinal peristalsis and fluid loss, as well as nausea and vomiting: Enterotoxins
Superantigens stimulates proliferation of T cells and release of cytokines, produces leakage or cellular destruction of endothelial cells: Toxic Shock Syndrome
Converts fibrinogen to fibrin: Coagulase
Catalyses removal or hydrogen peroxide: Catalase
Hydrolyzed in connective tissue, promoting the spread of staphylococci in tissue: Hyaluronidase

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