juxukuse's version from 2016-09-28 01:52

Section 1

Question Answer
reduction of total no. of rbc, in circulating blood quality or quantity of hemoglobinanemia
someone with anemia will have whata reduced oxygen-carrying capacity
symptoms of anemiafaigue, weakness dyspnea pallor tachycardia inflammed d. tract stomatits
more sever anemiachest pain, heart failure and syncope, a type of shock
most common type of anemia worldwideiron defeceincy anemia (microcytic-hypochromic anemia) in women mostly
progression of iron deficiency anemia causesbrittle thin coarsely ridged and spoon shaped nails, red sore painful tongue

Section 2

Question Answer
caused by a lack of intrinsic factor from the gastric parietal cellspernicious anemia (vitamin B12 deficiency)
what happens to make the person get pernicious anemiaantibodies against IF
what is needed for vitamin B12 absorptionIntrinsic factor
what does vitamin B12 deficiency lead toimpaired maturation of rbc
failure of bone marrow leading to loss of stem cells pancytopeniaAplastic Anemia
pancytopenia refers to decreased erythrocytes, leukocytes and platelets (lots of things decreased)

Section 3

Question Answer
what is the causes of aplastic anemiaradiation, chemo Dilantin (seizure med) sometimes unknown
If a person with Aplastic Anemia is a risk for Chemo stem cells are harvested before treatment
what else may play a cause in Aplastic Anemiaviruses and genetics
Pallor weakness dyspnea leukopenia with recurrent infections thromboycytopeniaS/S of Aplastic Anemia
with aplastic anemia thrombocytopenia looks like what petechiae (flat red pinpoint hemorrhages on the skin)
remove any bone marrow suppressants, bone marrow transplanttreatment for Aplastic Anemia (Treat the cause)

Section 4

Question Answer
excessive destruction of RBC's or hemolysis leading to low eryth. count & low hemog.Hemolytic Anemia
causes of Hemolytic Anemiagentetics, infections transfusion reactions blood incompatibilities, autoimmune
what is an example of a Hemolytic AnemiaSickle Cell anemia
overproduction of RBC's polycythemia
increase in RBC's Erythrocytosis
how would polycythemia lookincreased blood viscosity, distended blood vessels, sluggish flow
with sluggish flow of blood because of polycythemia what happensfrequent thromboses and infarction in body, spleen and liver are congested (plugged up

Section 5

Question Answer
considedred a neoplastic disorder (cancer)polycythemia
how is polycythemia treatedperiodic phlebotomy
thrombocytopenia, from acute viral infection autoimmune reaction (hiv), chemocauses of excessive bleeding (Blood Clotting Disorders)
radiation end stage renal failure, vitamin K defieceincy, liver disease genetics coumadinMORE reasons of (blood clotting disorders)
bleeding gums, bosebleeds, eccymosis bleeding into joint hemoptysis hematemisisS/S of blood clotting disorder
how would the stools of a person with blood clotting disorder looktarry stools
hemoptysiscoughing up blood

Section 6

Question Answer
deficit in clotting factor 8hemophilia
S/S of hemophiliasevere hemorrhage after minor tissue trauma, persistent oozing hemorrhage into joints
hemarthrosis and hematuria meanshemorrhage into joints or blood in urine
how is hemophilia treatedreplacement of factor 8
Acquired disorder where clotting and hemorrhage simultaneously occurDIC
DIC is the result of what Increased activation of clotting procces in Microcirculation of the body
after the increased activation of clotting process in Microcirculation of bodythen accelerated fibrinolysis (breaks down the clots)

Section 7

Question Answer
where does the clots occur in DICMicrocirculation of the body
can DIC be inheritedno it is acquired
how does the bleeding occurfibrin split products are made and they break down the clots in DIC
primary initiator of DICendothelial damage
infections-sepsis, hematologic cancers complications of pregnanceother causes of DIC
treatment for DICremove the stumulus, blood replacement, (heparin)

Section 8

Question Answer
malignant disorder os the WBC's Leukemia
Undifferentiated, immature, nonfunctional cells that multiply uncontrollablyLeukemia disease
where does the cells in leukemia multiple atthe bone marrow, then released in general circulation
what organs do leukemia infiltratelymph nodes, spleen, liver and brain
how is leukemia in childrenacute
how is leudemia in adultschronic

Section 9

Question Answer
Infection, Anemia, bleeding purpura, petechiae, eccymosis, thrombosis, hemorrhageS/S of leukemia
more S/S of leukemiaDIC, weight loss, bone pain, elevated uric acid
how woud the liver speen and lymph nodes look in leukemiaenlarged
enlarged lymph nodes palpable and tenderlymphadenopathy
drainage of an inflammatory lesion located near the enlared nodelocal lymphadenopathy
occurs in the presence of malignant or nonmalignant diseasegeneral lymphadenopathy

Section 10

Question Answer
malignant transformation of a lymphocyte and proliferation in the lymph nodesmalignant lymphomas
two major categories of malignant lymphomashodgkins lymphoma and non hodgkins lymphoma
initially affect lymph node in the neck and then later spreads to othershodgkins lymphoma
which antibodies are affected in hodgins lymphomaT lymohocytes
what is hodkins lymphoma characterized bypresence of Reed Sternberg cells in the lymph nodes
cells necessary for diagnosis of hodgkins lymphomaReed Sternberg Cells

Section 11

Question Answer
enlarged painless lymph nodes in neck 1st sign usuallyhodgkins lymphoma
other signs of hodgkins lymphomamediastinal mass, splenomegaly, and abdominal mass
what other signs will the person with hodgkins lymphoma havefever, weight loss, night sweats, pruritus, anemia, recurring infection
incedence increasing due to HIV infectionnon-hodgkins lymphoma
linked to chromosome translocationsnon-hodgkins lymphoma disease
what kind of infections with nonhodgkins lymphomaviral and bacterial, environmental agents, immunodeficiency and autoimmune disorder
what is often present at diagnosismultiple node involvement
what is the survival rate in non hodkins lymphomalower survival rate than hodgkins lymphoma but seeing some improvement

Section 12

Question Answer
most common type of non hodkins lymphoma in childrenBurkitt Lymphoma
Burkitt LymphomaVery fast growing tumor of the jaw and facial bones
what is found in nasopharyngeal secretions of patients with burkett lymphomaEpstein Barr virus
B cell cancerMultiple Myeloma
multiple myelomainfiltrate bone marrow and aggregate into tumor masses in skeketal system
which proteins are abnormal in multiple myelomaM Proteins
how would someone with multiple myeloma look besides pathological fracturesHypercalcemia, renal failure (bence Jones protein), bone lesions